Dysfunction of the Voltage-Gated K Channel b2 Subunit in a Familial Case of Brugada Syndrome

نویسندگان

  • Vincent Portero
  • Floriane Simonet
  • Gildas Loussouarn
  • Flavien Charpentier
چکیده

Vincent Portero, PhD;* Solena Le Scouarnec, PhD;* Zeineb Es-Salah-Lamoureux, PhD;* Sophie Burel, MS; Jean-Baptiste Gourraud, MD, PhD; St ephanie Bonnaud, PhD; Pierre Lindenbaum, PhD; Floriane Simonet, MS; Jade Violleau, BS; Estelle Baron, BS; El eonore Moreau, MS; Carol Scott, MS; St ephanie Chatel, PhD; Gildas Loussouarn, PhD; Thomas O’Hara, PhD; Philippe Mabo, MD; Christian Dina, PhD; Herv e Le Marec, MD, PhD; Jean-Jacques Schott, PhD; Vincent Probst, MD, PhD; Isabelle Bar o, PhD; C eline Marionneau, PhD; Flavien Charpentier, PhD; Richard Redon, PhD;

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Dysfunction of the Voltage‐Gated K+ Channel β2 Subunit in a Familial Case of Brugada Syndrome

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SCN5A (sodium channel, voltage-gated, type V, alpha subunit) gene encodes the cardiac sodium channel, a member of the voltage-gated sodium channel family. SCN5A mutations have been associated with a variety of inherited arrhythmias, including long QT syndrome and Brugada syndrome. We report an autopsy case of sudden unexpected nocturnal death syndrome. A man in his thirties died at night while ...

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INTRODUCTION Brugada syndrome is a hereditary arrhythmia characterized by a specific electrocardiographic pattern and an increased risk of sudden cardiac death, with an apparent absence of structural abnormalities or ischemic heart disease. To date, mutations in the sodium channel, voltage-gated, type V, alpha subunit gene and glycerol-3-phosphate dehydrogenase 1-like gene are estimated to acco...

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تاریخ انتشار 2016